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| 1 | Fixed point theorems for weak contractions in cone metric spaces显示文摘 | Fatma A S | 2010 | Int Journal of Math Analysis2010，7，48： | 1 |
| 2 | Th2 cytokine-induced up- regulation of 1 l beta-hydroxysteroid dehydrogenase-1 facil- itates glucocorticoid suppression of proasthmatic airway smooth muscle function显示文摘 | HU A FATMA S CAO J | 2009 | Am J Physiol Lung Cell Mol Physiol2009，296，5： | 1 |
| 3 | Mechanistic in- vestigation of the anodic oxidation of 3,4,5-trimethoxy- toluene in acetonitrile 显示文摘 | Ayoub H S Fatma M M Christian A | 2002 | J Electroanal Chem2002，537，： | 1 |
| 4 | Mechanistic in- vestigation of the anodic oxidation of p-methoxytoluene in dry and wet acetonitrile 显示文摘 | Ayoub H S Fatma M M Christian A | 1999 | J Electroanal Chem1999，464，： | 1 |
| 5 | Assessment of hepatic fibrosis in pediatric cases with hepatitis C virus in Egypt显示文摘AIM: To assess hepatic fibrosis and factors associated with its progression in children with HCV infection. METHODS: At the Hepatology Unit,Cairo University Children's Hospital,a single liver biopsy was performed to 43 children with HCV infection after an informed consent between 1998-2004. Their mean age at liver biopsy was 8.67 ± 4.3 years. RESULTS: Among the 43 patients' biopsies,12 (27.9%) were having no fibrosis,20 (46.5%) mild fibrosis and 11 (25.6%) moderate to severe fibrosis. The median time for development of fibrosis was estimated to be 5.5 years. Developing fibrosis was significantly associated with shorter duration from first detected ALT elevation to biopsy (12 mo vs 1.2 mo,P = 0.015) and having higher levels of direct serum bilirubin (0.3 mg/dL vs 0.5 mg/dL,P = 0.048). No association was found between fibrosis stage and the presence of co-morbid conditions (P = 0.33). CONCLUSION: Hepatic fibrosis was present in 72.1% of children with HCV infection. The development of fibrosis was associated with higher levels of direct serum bilirubin. There was no significant association between fibrosis and age,duration of infection,risk factors,co-morbid conditions and most biochemical parameters. | Manal A El-Hawary Mona S El-Raziky Gamal Esmat Hanan Soliman Amr Abouzied Maissa El-Raziky Wafaa El-Akel Rokaya El-Sayed Fatma Shebl Abdel Aziz Shaheen Hanaa El-Karaksy | 2007 | World Journal of Gastroenterology2007，13，20： | 1 |
| 6 | 显示文摘 | Ansari S A Nisar A Fatma B | 2012 | Materials Science and Engineering B-Advanced Functional ~lid State Materials2012，177，5： | 1 |
| 7 | HLA-DQB1 * 等位基因和基因危险性到类型 1 糖尿病 mellitus显示文摘 AIM: To determine human leukocyte antigen (HLA)DQB1 allele association with susceptibility to type 1 diabetes (T1D) and to clinical and laboratory findings. METHODS: This study was conducted on 85 unrelated Egyptian children with T1D recruited consecutively from the Pediatric Diabetes Endocrinology outpatients Clinic; Mansoura University Children’s Hospital, Egypt. Patient mean follow up period was 2.5 years. Patients were subdivided according to level of HbA1c (optimal/suboptimal control < 8.5% and poor control ≥ 8.5%). Thecontrol group consisted of 113 unrelated ageand sexmatched healthy subjects without T1D or other autoimmune diseases. Genomic DNA extraction was done for all subjects using a DNA isolation kit. HLA-Class Ⅱ-DQB1 allele typing was carried out with a polymerase chain reaction-sequence-specific oligonucleotide probe using a INNO-LiPA HLA-DQB1 update kit. RESULTS: Significant differences were detected between Egyptian patients with T1D and control groups in the frequencies of DQB1*02 [44.4% vs 18.6%, corrected P value (Pc) < 0.001] and DQB1*03 (41.2% vs 24.4%, Pc < 0.001). Significant differences were also observed between control groups and T1D patients in the frequencies of DQB1*05 (14.6% vs 7.2%, P = 0.029) and DQB1*06 (34.1% vs 7.2%, P < 0.001). However, after correction for multiple comparisons, the significance was retained for HLA-DQB1*06 (Pc < 0.001) but lost for HLA-DQB1*05. HLA-DQB1*0201, *0202, *030201 were positively associated with T1D (Pc = 0.014, Pc < 0.001, and Pc < 0.001 respectively), while HLA-DQB1*060101 was negatively associated (Pc < 0.001) with the condition. Although the HLA-DQB1 alleles 030101 and 050101 were significantly higher in controls (P = 0.016, P = 0.025 respectively), both of them lost statistical significance after correction of P value. The frequency of the HLA-DQB1 genotypes 02/02, 02/03, and 03/03 was higher in T1D patients, and the frequency of the genotypes 03/06, 05/06, and 06/06 was higher in controls, these differences being statistically significant before correction. After correction, the genotypes 02/02, 02/03 in T1D, and the genotypes 03/06, 06/06 in controls were still significant (Pc = 0.01, Pc < 0.001, Pc < 0.001, and Pc = 0.04, respectively). Non-significant associations were found between the frequency HLA-DQB1 alleles and genotypes in T1D in relation to the grade of diabetic control, Microalbuminuria, age, gender, age of presentation, weight, height, frequency of diabetic ketoacidosis (P =0.42), serum cholesterol, and fasting and post-prandial level of C-peptide (P = 0.83, P = 0.9, respectively). CONCLUSION: The Current work suggests that HLADQB1 alleles *030201, *0202, *0201, and genotypes 02/03, 02/02 may be susceptibility risk factors for development of T1D in Egyptian children, while the HLADQB1*060101 allele, and 03/06, 06/06 genotypes may be protective factors. HLA-DQB1 alleles and genotypes do not contribute to microalbuminuria or grade of diabetic control. | Youssef M Mosaad Fatma A Auf Shereen S Metwally Ashraf A Elsharkawy Amany K El-Hawary Rasha H Hassan Ziyad E Tawhid Farha A El-Chennawi | 2012 | World Journal of Diabetes2012，3，8： | 1 |
| 8 | Using Objective Structured Clinical Examination (OSCE) in Undergraduate Psychiatric Nursing Education: Is It Reliable and Valid?显示文摘 | Abeer A S Fatma H R Mervat M E | 2012 | Nurse Edue Today2012，32，3： | 1 |
| 9 | Propofol offers no advantage over isoflurane anesthesia for cerebral protection during cardiopulmonary bypass: a preliminary study of S - 100β protein levels显示文摘 | MERAL K FATMA S ALEV A | 2004 | Can J Anaesth2004，51，7： | 1 |
| 10 | Impact:A platform for collaborating agents显示文摘 | A A Khaled O Fatma & V S Subrahmanian | 1999 | IEEE Intelligent Systems (March/April)1999，，： | 1 |
| 11 | Identity, regulation and activity of inducible diterpenoid phytoalexins in maize显示文摘 | ERIC A S FATMA K ALISA H | 2011 | PNAS2011，108，13： | 1 |
| 12 | Association between tumor necrosis factor-alpha gene promoter polymorphism at position -308 and acne in Turkish patients显示文摘 | K?ymet Baz M. Emin Erdal Ay?a Cordan Yaz?c? Fatma S?ylemez Ula? Güven? Bahar Ta?delen Güliz Ikizo?lu | 2008 | Archives of Dermatological Research2008，，7： | 1 |
| 13 | Dissoltion of Lignites in Tetralin at Ambient Temperature: Effects of Ultraviolet Tradiation显示文摘 | Fatma S Olcay A | 1998 | FuelProcessing Technology1998，55，： | 1 |
| 14 | Vascular endothelial growth factor,p53,and the H-ras oncogene in Egyptian patients with bladder cancer显示文摘AIM:To evaluate the relationship between vascular endothelial growth factor(VEGF),p53,and the H-ras oncogene and different clinicopathological parameters in Egyptian patients with Schistosoma-associated transitional cell carcinoma of the bladder.METHODS:The study included 50 patients with transitional cell carcinoma for whom radical cystectomy and urinary diversions were carried out.VEGF and p53 protein expressions were evaluated with an immunohistochemical staining method,and H-ras oncogene mutations were analyzed with a polymerase chain reaction-restriction fragment length polymorphism(PCR-RFLP) technique.RESULTS:High grade tumors revealed higher p53 immunostaining than low grade tumors(P = 0.016).p53 and VEGF protein expressions,as well as H-ras oncogene mutations,had an insignificant impact on patient outcomes(P = 0.962,P = 0.791,and P = 967,respectively).Cancer extension to regional lymph nodes was associated with poor outcomes(P = 0.008).CONCLUSION:VEGF,p53 and the H-ras oncogene have no relation to patient survival and outcome in Schistosoma-associated transitional cell carcinoma. | Farha A El-Chennawi Fatma A Auf Shereen S Metwally Youssef M Mosaad Atallah A Shaaban Mahmoud Abdo El-Baz Ziyad E Tawhid Zakaria F Lotfy | 2009 | World Journal of Gastrointestinal Oncology2009，1，1： | 0 |
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