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| 1 | Unintentional forking analysis in wireless blockchain networks显示文摘The forking problem plays a key role in the security issue,which is a major concern in the blockchain system.Although many works studied the attack strategy,consensus mechanism,privacy-protecting and security performance analysis,most of them only address the intentional forking caused by a malicious attacker.In fact,without any attacker,unintentional forking still remains due to transmission delay and failure,especially in wireless network scenarios.To this end,this paper investigates the reason for generating unintentional forking and derives the forking probability expression in Wireless Blockchain Networks(WBN).Furthermore,in order to illustrate the unintentional forking on the blockchain system,the performances in terms of resource utilization rate,block generation time,and Transaction Per Second(TPS)are investigated.The numerical results show that the target difficulty of hash algorithm in generating a new block,the delay time of broadcasting,the network scale,and the transmission failure probability would affect the unintentional forking probability significantly,which can provide a reliable basis for avoiding forking to save resource consumption and improving system performance. | Qilie Liu Yinyi Xu Bin Cao Lei Zhang Mugen Peng | 2021 | Digital Communications and Networks2021，7，3： | 3 |
| 2 | Identification and molecular characterization of two novel mutations in COL1A2 in two Chinese families with osteogenesis imperfecta显示文摘成骨 imperfecta (OI,也已知的同样易碎的骨头疾病) 主要被变化在二种类型引起我编码类型的 pro-1 (I) 和 pro-2 (I) 链的骨胶原基因, COL1A1 和 COL1A2 我骨胶原分别地。有正染色体的主导的 OI 的二个中国家庭被识别并且描绘。连接分析在染色体 7q21.3-q22.1 上揭示了两个家庭的连接到 COL1A2。Mutational 分析用直接 DNA 顺序分析被执行。二个新奇错误感觉变化, c.3350A > G 和 c.3305G > C,在 exon 被识别在二个家庭的 49 COL1A2 分别地。c.3305G > C 变化在 codon 由丙氨酸残余(A) 导致了 glycine 残余(G) 的替换 1102 (p.G1102A ) ,它被发现在另外的家庭被变异进丝氨酸(S) , argine (R) ,丁氨二酸酸(D) ,或缬氨酸(V) 。c.3350A > G 变体可以是导致 p.Y1117C 的一个 de novo 变化。两个变化在各自的家庭与 OI 共同分离,并且没在 100 正常控制被发现。G1102 和 Y1117 残余高度 evolutionarily 从 zebrafish 被保存到人。Mutational 分析没在 COX-2 基因(OI 的修饰词基因) 识别任何变化。这研究识别引起 OI 的二新奇变化 p.G1102A 和 p.Y1117C,显著地扩展引起 OI 的 COL1A2 变化的光谱,并且有在 OI 的出生前的诊断的一个重要含意。 | Zhenping Xu Yulei Li Xiangyang Zhang Fanming Zeng Mingxiong Yuan Mugen Liu Qing Kenneth Wang Jing Yu Liu | 2011 | Journal of Genetics and Genomics2011，38，4： | 3 |
| 3 | Channel Estimation for Reconfigurable Intelligent Surface Assisted Wireless Communication Systems in Mobility Scenarios显示文摘Reconfigurable intelligent surface(RIS)can manipulate the wireless propagation environment by smartly adjusting the amplitude/phase in a programmable panel,enjoying the improved performance.The accurate acquisition of the instantaneous channel state information(CSI)in the cascaded RIS chain makes an indispensable contribution to the performance gains.However,it is quite challenging to estimate the CSI in a time-variant scenario due to the limited signal processing capability of the passive elements embedded in a RIS pannel.In this work,a channel estimation scheme for the RIS-assisted wireless communication system is proposed,which is demonstrated to perform well in a time-variant scenario.The cascaded RIS channel is modeled as a state-space model based upon the mobility situations.In addition,to fully exploit the time correlation of channel,Kalman filter is employed by taking the prior information of channels into account.Further,the optimal reflection coefficients are derived according to the minimum mean square error(MMSE)criterion.Numerical results show that the proposed methods exhibit superior performance if compared with a conventional channel estimation scheme. | Zhendong Mao Mugen Peng Xiqing Liu | 2021 | China Communications2021，18，3： | 3 |
| 4 | Novel ACTG1 mutation causing autosomal dominant non-syndromic hearing impairment in a Chinese family显示文摘The γ-actin (ACTG1) gene is a cytoplasmic nonmuscle actin gene, which encodes a major cytoskeletal protein in the sensory hair cells of the cochlea. Mutations in ACTG1 were found to cause autosomal dominant, progressive, sensorineural hearing loss linked to the DFNA 20/26 locus on chromosome 17q25.3 in European and American families, respectively. In this study, a novel missense mutation (c.364A>G; p.I122V) co-segregated with the affected individuals in the family and did not exist in the unaffected family members and 150 unrelated normal controls. The alteration of residue Ile122 was predicted to damage its interaction with actin-binding proteins, which may cause disruption of hair cell organization and function. These findings strongly suggested that the I122V mutation in ACTG1 caused autosomal dominant non-syndromic hearing impairment in a Chinese family and expanded the spectrum of ACTG1 mutations causing hearing loss. | Ping Liu Hu Li Xiang Ren Haiyan Mao Qihui Zhu Zhengfeng Zhu Rong Yang Wenlin Yuan Jingyu Liu Qing Wang Mugen Liu | 2008 | Journal of Genetics and Genomics2008，35，9： | 3 |
| 5 | Intent-based networks for 6G:Insights and challenges显示文摘Intent-Based Networks(IBNs),which are originally proposed to introduce Artificial Intelligence(AI)into the sixth-generation(6G)wireless networks,can effectively solve the challenges of traditional networks in terms of efficiency,flexibility,and security.IBNs are mainly used to transform users’business intent into network configuration,operation,and maintenance strategies,which are prominent for designing the AI-enabled 6G networks.In particular,in order to meet the massive,intelligent service demands and overcome the time-varying radio propagation,IBNs can continuously learn and adapt to the time-varying network environment based on the massive collected network data in real-time.From the aspects of both the core network and radio access network,this article comprehensively surveys the architectures and key techniques of IBNs for 6G.In particular,the demonstration platforms of IBNs,such as the Apstra Operating System,Forward Networks Verification Platform,and One Convergence Service Interaction Platform,are presented.Moreover,the industrial development of IBNs is elaborated,including the emerging new products and startups to solve the problems of open data platforms,automated network operations,and preemptive network fault diagnosis.Finally,several open issues and challenges are identified as well to spur future researches. | Yiming Wei Mugen Peng Yaqiong Liu | 2020 | Digital Communications and Networks2020，6，3： | 3 |
| 6 | The R1947X mutation of NF1 causing autosomal dominant neurofibromatosis type 1 in a Chinese family显示文摘Neurofibromatosis type 1 is a common autosomal dominant disorder with a high rate of penetrance. It is caused by the mutation of the tumor suppressor gene NF1, which encodes neurofibromin. The main function of neurofibromin is down-regulating the biological activity of the proto-oncoprotein Ras by acting as a Ras-specific GTPase activating protein. In this study, we identified a Chinese family affected with neurofibromatosis type 1. The known gene NF1 associated with NF1 was studied by linkage analysis and by direct sequencing of the entire coding region and exon-intron boundaries of the NF1 gene. The R1947X mutation of NF1 was identified, which was co-segregated with affected individuals in the Chinese family, but not present in unaffected family members. This is the first report, which states that the R1947X mutation of NF1 may be one of reasons for neurofibromatosis type 1 in Chinese population. | Qinbo Yang Changzheng Huang Xiaoying Yang Yinfu Feng Qing Wang Mugen Liu | 2008 | Journal of Genetics and Genomics2008，35，2： | 2 |
| 7 | A comparison of ophthalmic education in China and America显示文摘Training qualified ophthalmic professional is crucial for any eye care system worldwide.Education of modern western Ophthalmology in China started late but develops rapidly.This review focused on ophthalmic education in China and US,describing details of the programs and analyzing the differences.This summary may provide useful information for practitioners of medical education from both countries and help improve the present training designs. | Ziyuan Liu Mugen Liu Robert Chang Ping Huang Chun Zhang | 2017 | Annals of Eye Science2017，，1： | 1 |
| 8 | T14484C and T14502C in the mitochondrial ND6 gene are associated with Leber’s hereditary optic neuropathy in a Chinese family显示文摘 | Shirong Zhang Lejin Wang Yansheng Hao Pengyun Wang Ping Hao Ke Yin Qing K Wang Mugen Liu | 2008 | Mitochondrion2008，，3： | 1 |
| 9 | Software defined intelligent satellite-terrestrial integrated networks:Insights and challenges显示文摘Satellite-Terrestrial integrated Networks(STNs)have been advocated by both academia and industry as a promising network paradigm to achieve service continuity and ubiquity.However,STNs suffer from problems including poor flexibility of network architecture,low adaptability to dynamic environments,the lack of network intelligence,and low resource utilization.To handle these challenges,a Software defined Intelligent STN(SISTN)architecture is introduced.Specifically,the hierarchical architecture of the proposal is described and a distributed deployment scheme for SISTNs controllers is proposed to realize agile and effective network management and control.Moreover,three use cases in SISTNs are discussed.Meanwhile,key techniques and their corresponding solutions are presented,followed by the identification of several open issues in SISTNs including compatibility with existing networks,the tradeoff between network flexibility and performance,and so on. | Shuo Yuan Mugen Peng Yaohua Sun Xiqing Liu | 2023 | Digital Communications and Networks2023，9，6： | 0 |
| 10 | Functional characterization of novel NPRL3 mutations identified in three families with focal epilepsy显示文摘Focal epilepsy accounts for 60% of all forms of epilepsy, but the pathogenic mechanism is not well understood. In this study,three novel mutations in NPRL3(nitrogen permease regulator-like 3), c.937_945del, c.1514dup C and 6,706-bp genomic DNA(g DNA) deletion, were identified in three families with focal epilepsy by linkage analysis, whole exome sequencing(WES) and Sanger sequencing. NPRL3 protein is a component of the GATOR1 complex, a major inhibitor of m TOR signaling. These mutations led to truncation of the NPRL3 protein and hampered the binding between NPRL3 and DEPDC5, which is another component of the GATOR1 complex. Consequently, the mutant proteins enhanced m TOR signaling in cultured cells, possibly due to impaired inhibition of m TORC1 by GATOR1. Knockdown of nprl3 in Drosophila resulted in epilepsy-like behavior and abnormal synaptic development. Taken together, these findings expand the genotypic spectrum of NPRL3-associated focal epilepsy and provide further insight into how NPRL3 mutations lead to epilepsy. | Shiyue Du Sheng Zeng Li Song Hongying Ma Rui Chen Junyu Luo Xu Wang Tingbin Ma Xuan Xu Hao Sun Ping Yi Jifeng Guo Yaling Huang Mugen Liu Tao Wang Wei-Ping Liao Luoying Zhang Jing Yu Liu Beisha Tang | 2023 | Science China(Life Sciences)2023，66，9： | 0 |
| 11 | Mechanisms of PiT2-loop7 Missense Mutations Induced Pi Dyshomeostasis显示文摘PiT2 is an inorganic phosphate(Pi)transporter whose mutations are linked to primary familial brain calcification(PFBC).PiT2 mainly consists of two ProDom(PD)domains and a large intracellular loop region(loop7).The PD domains are crucial for the Pi transport,but the role of PiT2-loop7 remains unclear.In PFBC patients,mutations in PiT2-loop7 are mainly nonsense or frameshift mutations that probably cause PFBC due to C-PD1131 deletion.To date,six missense mutations have been identified in PiT2-loop7;however,the mechanisms by which these mutations cause PFBC are poorly understood.Here,we found that the p.T390A and p.S434W mutations in PiT2-loop7 decreased the Pi transport activity and cell surface levels of PiT2.Furthermore,we showed that these two mutations attenuated its membrane localization by affecting adenosine monophosphate-activated protein kinase(AMPK)-or protein kinase B(AKT)-mediated PiT2 phosphorylation.In contrast,the p.S121C and p.S601W mutations in the PD domains did not affect PiT2 phosphorylation but rather impaired its substrate-binding abilities.These results suggested that missense mutations in PiT2-loop7 can cause Pi dyshomeostasis by affecting the phosphorylation-regulated cell-surface localization of PiT2.This study helps understand the pathogenesis of PFBC caused by PiT2-loop7 missense mutations and indicates that increasing the phosphorylation levels of PiT2-loop7 could be a promising strategy for developing PFBC therapies. | Hao Sun Xuan Xu Junyu Luo Tingbin Ma Jiaming Cui Mugen Liu Bo Xiong Shujia Zhu Jing-Yu Liu | 2023 | Neuroscience Bulletin2023，39，1： | 0 |
| 12 | Recent Advances of Simultaneous Wireless Information and Power Transfer in Cellular Networks显示文摘As a promising solution to alleviating the energy bottleneck in wireless devices with limited battery capacity,simultaneous wireless information and power transfer(SWIPT)techniques have been widely researched in cellular networks.To further improve the spectral and energy efficiency of wireless information and power transfer,the combination of SWIPT and new techniques in cellular networks has drawn much attention recently.In this paper,we comprehensively survey the key techniques for SWIPT,the combination of SWIPT and new techniques in cellular networks,challenges and open issues.The key techniques for SWIPT including traditional power splitting,time switching,etc.,and joint receiving and transmitting techniques such as eigenchannels and mixed signals are provided in detail.Furthermore,the applications of SWIPT to recent techniques such as SWIPT-assisted non-orthogonal multiple access,SWIPT-assisted device-to-device communication,and SWIPT-assisted full-duplex communication,are comprehensively summarized in this paper.The potential open issues including the management of dynamic harvested energy,trading between wireless power transfer and traffic offloading,and effects of the mode switching at energy harvesting devices,are outlined as well. | LIU Binghong PENG Mugen ZHOU Zheng | 2018 | ZTE Communications2018，16，1： | 0 |
| 13 | Performance analysis of diffusion-based decode-and-forward relay with depleted molecule shift keying显示文摘The distance-decay effect of molecular signals makes communication range a major challenge for diffusion-based Molecular Communication(MC).To solve this problem,the intermediate nano-machine is deployed as a relay between the transmitter and its intended receiver nano-machines.In this work,we employ the Depleted Molecule Shift Keying(D-MoSK)to model a Decode-and-Forward(DF)relay communication scheme.The closed-form expression of Bit Error Rate(BER)for the concerned DF relay with D-MoSK is derived.Meanwhile,the maximum a posteriori probability,minimum error probability,and maximum likelihood schemes are formulated for data detection.The relationships between BER and other key parameters,including the number of released molecules,receiving radius,and relay position,are investigated in detail.Simulation results show that the proposed scheme can improve communication reliability significantly.Moreover,the performance gain can be maximized by optimizing the position of the relay and the receiving radius. | Jiaxing Wang Mugen Peng Yaqiong Liu | 2021 | Digital Communications and Networks2021，7，3： | 0 |
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