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33篇 您的检索式:作者名="McCurrach ME"
    题名 作者 年代 出处 被引量
1Molecular basis of myotonic dystrophy:expansion of a trinucleotide (CTG) repeat at the 3-prime end of a transcript encoding a protein kinase family member显示文摘Brook JD McCurrach ME Harley HG 1992Cell1992,68,:1
2FADD:essential for embryo development and signaling from some,but not all,inducers of apoptosis显示文摘Yeh WC Pompa JL McCurrach ME 1998Science1998,279,5358:1
3Oncogenic ras provokes premature cell senescence associated with accumula tion of p53 and p16ink4a显示文摘Serrano M Lin AW McCurrach ME 1997Cell1997,88,5:1
4INK4a/ARF mutations accelerate lymphomagenesis and promote chemoresistance by disabling p53显示文摘Schmitt CA McCurrach ME de Stanchina E 1999Genes Dev1999,13,:1
5FADD:Essential for embryo development and signaling from some,but not all,induces of apoptosis显示文摘Yeh WC Pompa JLDI McCurrach ME 1998Science1998,279,5358:1
6Bax-deficiency promotes drug resistance and oncogenic transformation by attenuating p53-dependent apotosis显示文摘McCurrach ME Connor TMF Knudson CM 1997Proc Natl Acad Sci USA1997,94,:1
7Ink4α/ARF mutations accelerate lymphomagenesis and promote chemoresistance by disabling p53 显示文摘Schmitt CA Mccurrach ME Stanchina E 1999Genes Dev1999,13,20:1
8Bax-deficiency promotes drug resistance and oncogenic transformation by attenuating p53 dependent apoptosis显示文摘McCurrach ME Connor TM Knudson CM 1997Proc Natl Acad Sci USA1997,94,6:1
9INK4a/ARF mutations accelerate lymphomagenesis and promote chemoresistance by disabling p53 显示文摘Schmitt CA Mccurrach ME De Stanchina E 1999Gene Dev1999,13,20:1
10Oncogenic fas provokes premature cell senescence associated with accumulation of p53 and p16(INK4a)显示文摘Serrano M Lin AW McCurrach ME Beach D Lowe SW 0,,:1
11Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a显示文摘Serrano M Lin AW McCurrach ME 1997Cell1997,88,5:1
12Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a显示文摘Serrano M Lin AW McCurrach ME 1997Ce111997,88,5:1
13Oncogenic ras provokes premature cell senescence associated with accu- mulation of p53 and pl6INK4a 显示文摘Serrano M Lin AW McCurrach ME 1997Cell1997,88,5:1
14Oncogenic ras pro- vokes premature cell senescence associated with accumulation of p53 and p16INK4a显示文摘Serrano M Lin AW Mccurrach ME 1997Ce111997,88,5:1
15INK4a/ ARF mutations accelerate lymphomagenesis and promote che moresistance by disabling p53显示文摘Schmitt CA McCurrach ME De Stanchina E 1999Genes Dev1999,13,20:1
16Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a显示文摘Serrano M Lin AW Mccurrach ME 1997Cell1997,88,:1
17FADD:Essential for embryo development and signaling from some, but not all,inducers of apoptosis显示文摘 Pompa JLDI McCurrach ME 1998Sciense1998,279,:1
18Molecular basis of myotonic dystrophy:Expansion of a trinucleotide(CTG)repeat at the 3'end of a transcript encoding a protein kinase family member显示文摘Brook JD McCurrach ME Harley HG 1992Cell1992,68,:1
19Expansion of a CUG trinucleotide repeat in the 3' untranslated region of myotonic dystrophy protein kinase transcripts results in nuelear retention of transcripts显示文摘Davis BM McCurrach ME Taneja KL 1997Proc Natl Acad Sci U S A1997,94,14:1
20Oncogenic ras provokespremcUure cell senescence associated with accumulation of pS3 andpl6INK4a显示文摘Serrano M Lin AW McCurrach ME 1997Cell1997,88,5:1
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