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7篇 您的检索式：作者名="Kuenzi F"

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		题名	作者	年代	出处	被引量
	1	Age-related impairment of synaptic transmission but normal long-term potentiation in transgenic mice that overexpress the human APP695SWE mutant form of amyloid precursor protein显示文摘	Fitzjohn SM Morton RA Kuenzi F	2001	J Neurosci2001，21，：	1
	2	Age-related impairment of synaptic transmission but normal long-term potentiation in trans- genie mice that overexpress the human APP695SWE mutant form of amyloid precursor protein 显示文摘	Fitzjohn SM Morton RA Kuenzi F	2001	J Neurosci2001，21，：	1
	3	A study of long-term potentiation in transgenic mice over-expressing mutantforms of both amyloid precursor protein and presenilin-1显示文摘	Fitzjohn SM Kuenzi F Morton RA	2010	Mol Brain2010，3，1：	1
	4	Age-related impairment of synaptic transmission but normal long-term potentiation in transgenic mice that overexpress the human APP695SWE mutant form of amyloid precursor protein 显示文摘	Fitzjohn SM Morton RA Kuenzi F	2001	J Neurosci2001，21，13：	1
	5	Age-related impairment of synaptic transmission but normal long-term potentiation in transgenic mice that overexpress the human APP695SWE mutant form of amyloid precursor protein 显示文摘	Fitzjohn SM Morton RA Kuenzi F	2001	The Journal of Neuroscience2001，21，13：	1
	6	A study of long-term potentiation in transgenic mice over-expressing mutant forms of both amyloid precursor protein and presenilin-1显示文摘	Fitzjohn SM Kuenzi F Morton RA	2010	Mol Brain2010，3，1：	1
	7	A study of long-term potentiation in transgenic mice over-expressing mu-tant forms of both amyloid precursor protein and presenil-in-1显示文摘	Fitzjohn SM Kuenzi F Morton RA	2010	Mol Brain2010，3，1：	1

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