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1Macrophage migration inhibitory factor gene polymorphisms in inflammatory bowel disease: An association study in New Zealand Caucasians and meta-analysis显示文摘AIM:To investigate the association of macrophage migration inhibitory factor(MIF)promoter polymorphisms with inflammatory bowel disease(IBD)risk.METHODS:One thousand and six New Zealand Caucasian cases and 540 Caucasian controls were genotyped for the MIF SNP-173G>C(rs755622)and the repeat polymorphism CATT5-8(rs5844572)using a predesigned TaqMan SNP assay and capillary electrophoresis,respectively.Data were analysed for single site and haplotype association with IBD risk and phenotype.Meta-analysis was employed,to assess cumulative evidence of association of MIF-173G>C with IBD.All published genotype data for MIF-173G>C in IBD were identified using PubMed and subsequently searching the references of all PubMed-identified studies.Imputed genotypes for MIF-173G>C were generated from the Wellcome Trust Case Control Consortium(and National Institute of Diabetes and Digestive and Kidney Diseases).Separate meta-analyses were performed on Caucasian Crohn’s disease(CD)(3863 patients,6031controls),Caucasian ulcerative colitis(UC)(1260 patients,1987 controls),and East Asian UC(416 patients and 789 controls)datasets using the Mantel-Haenszel method.The New Zealand dataset had 93%power,and the meta-analyses had 100%power to detect an effect size of OR=1.40 atα=0.05,respectively.RESULTS:In our New Zealand dataset,single-site analysis found no evidence of association of MIF polymorphisms with overall risk of CD,UC,and IBD or disease phenotype(all P values>0.05).Haplotype analysis found the CATT5/-173C haplotype occurred at a higher frequency in New Zealand controls compared to IBD patients(0.6 vs 0.01;P=0.03,OR=0.22;95%CI:0.05-0.99),but this association did not survive bonferroni correction.Meta-analysis of our New Zealand MIF-173G>C data with data from seven additional Caucasian datasets using a random effects model found no association of MIF polymorphisms with CD,UC,or overall IBD.Similarly,meta-analysis of all published MIF-173G>C data from East Asian datasets(416UC patients,789 controls)found no association of this promoter polymorphism with UC.James D Falvey Robert W Bentley Tony R Merriman Mark B Hampton Murray L Barclay Richard B Gearry Rebecca L Roberts 2013World Journal of Gastroenterology2013,19,39:9
2Quality in the technical performance of colonoscopy and the continuous quality improvement process for colonoscopy: recommendations of the U.S. Multi-Society Task Force on Colorectal Cancer显示文摘Douglas K Rex John H Bond Sidney Winawer Theodore R Levin Randall W Burt David A Johnson Lynne M Kirk Scott Litlin David A Lieberman Jerome D Waye James Church John B Marshall Robert H Riddell 2002The American Journal of Gastroenterology2002,,6:3
34 口腔癌全球发病率评估显示文摘该文旨在精确估计全球口腔癌的发病率。方法:分析国际癌症研究机构GLOBOCAN数据库中2002年175个国家的口腔癌发病资料。包括人口学资料、每个国家的疾病发病率及新增病例数。结果:资料显示,2002年全球口腔癌新增病例为274289例,发病率最高的国家为新几内亚.发病率为42/100000。其次为所罗门群岛和马来西亚.发病率分别为35/100000和33/100000,发病率明显高于其他国家,并且男性高于女性。Gorham E D James W E Mohr S B 2007中国口腔颌面外科杂志2007,5,1:3
4Meeting report:a hard look at the state of enamel research显示文摘The Encouraging Novel Amelogenesis Models and Ex vivo cell Lines(ENAMEL) Development workshop was held on 23 June 2017 at the Bethesda headquarters of the National Institute of Dental and Craniofacial Research(NIDCR). Discussion topics included model organisms, stem cells/cell lines, and tissues/3 D cell culture/organoids. Scientists from a number of disciplines,representing institutions from across the United States, gathered to discuss advances in our understanding of enamel, as well as future directions for the field.ophir d klein olivier duverger wendy shaw rodrigo s lacruz derk joester janet moradian-oldak megan k pugach j timothy wright sarah e millar ashok b kulkarni john d bartlett thomas gh diekwisch pamela den besten james p simmer 2017International Journal of Oral Science2017,9,4:3
5DNA damage induced by chronic inflammation contributes to colon carcinogenesis in mice显示文摘Meira Lisiane B Bugni James M Green Stephanie L Lee Chung-Wei Pang Bo Borenshtein Diana Rickman Barry H Rogers Arlin B Moroski-Erkul Catherine A McFaline Jose L Schauer David B Dedon Peter C Fox James G Samson Leona D 2008Journal of Clinical Investigation2008,,7:2
6COI1:An Arabidopsis gene required for jasmonate-regulated defense and fertility显示文摘Xie D X Feys B F James S 1998Science1998,280,:2
7D-MELD risk capping improves post-transplant and overall mortality under markov microsimulation显示文摘AIM: To hypothesize that the product of calculated Model for End-Stage Liver Disease score excluding exception points and donor age(D-MELD) risk capping ± Rule 14 could improve post liver transplant and overall survival after listing.METHODS: Probabilities derived from the United Network for Organ Sharing database between 2002 and 2004 were used to simulate potential outcomes for all patients listed for transplantation. The Markov simula-tion was then modified by screening matches using a 1200 or 1600 D-MELD risk cap ± allowing transplants for Model for End-Stage Liver Disease(MELD) ≤ 14(Rule 14). The differential impact of the rule changes was assessed.RESULTS: The Markov simulation accurately reproduced overall and post transplant survival. A 1200 D-MELD risk cap improved post-transplant survival. Both the 1200 and 1600 risk caps improved overall survival for waitlisted patients. The addition of Rule 14 further improved post transplant and overall survival by redistribution of donor livers to recipients in higher MELD subgroups. The mechanism for improved overall and post-transplant survival after listing was due to shifting a larger percentage of transplants to the moderate MELD score subgroup(MELD 15-29) while also ensuring that high MELD recipients have livers of high quality to achieve excellent post transplant survival.CONCLUSION: A 1200 D-MELD risk cap + Rule 14 provided the greatest overall benefit primarily by focusing liver transplantation towards the moderate MELD recipient.Jeffrey B Halldorson Robert L Carithers Jr Renuka Bhattacharya Ramasamy Bakthavatsalam Iris W Liou Andre A Dick Jorge D Reyes James D Perkins 2014World Journal of Transplantation2014,4,3:2
8Security Models for Web-based Applications 显示文摘James B D Joshi Walid G Aref 2001Communications of the ACM2001,44,2:2
9Complete genomes of two clinical Staphylococcus aureus strains:evidence for the rapid evolution of virulence and drug resistance显示文摘Holden MT Feil EJ Lindsay JA Peacock SJ Day NP Enright MC Foster TJ Moore CE Hurst L Atkin R Barron A Bason N Bentley SD Chillingworth C Chillingworth T Churcher C Clark L Corton C Cronin A Doggett J Dowd L Feltwell T Hance Z Harris B Hauser H Holroyd S Jagels K James KD Lennard N Line A Mayes R Moule S Mungall K Ormond D Quail MA Rabbinowitsch E Rutherford K Sanders M Sharp S Simmonds M Stevens K Whitehead S Barrell BG Spratt BG Parkhill J 2004Proc Natl Acad Sci USA2004,101,26:1
10Precision determination of lattice parameters and coefficients of thermal expansion of BiFeO3显示文摘Bucci J D Robertson B K James W J 1972J Appl Cryst1972,5,:1
11Uncertain envi- ronmental variables in GIS 显示文摘Gerard B M Heuvelink James D B 2008Encyclopedia of GIS2008,,24:1
12Growth and Magnetic Properties of MnO2-β Nanowire Microspheres显示文摘YANG J B ZHOU X D JAMES W J 2004Appl Phys Lett2004,85,15:1
13Dy-namic loads on transmission line structures due to gal-loping conductors 显示文摘Baenziger M A James W D Wouters B 1994IEEE Transactions on PowerDelivery1994,9,1:1
14Colo- rectal cancer screening显示文摘RANDALL W B JAMES S B KELLI B D 2010J Natl Compr Cane Netw2010,8,:1
15Body burden measurements and models to assess inhalation exposure to methyl tertiary butyl ether (MTBE)显示文摘Timothy J B James D P David A 1997Air and Waste Manage1997,47,7:1
16High-level expression of the phenylalanine ammonia lyase-encoding gene from Rhodosporidium toruloides in Sacchpromyces cerevisiae and Escherichia coli using a bifunctional expression system显示文摘JAMES D B FAULKNERA T MICK F T 1994Gene1994,143,:1
17, Tunable Narrow-Band Filter for LWIR Hyperspectral Imaging 显示文摘James T D Andrew B William S 2000Proceedings of SPIE2000,3948,:1
18The dimensionality of the maslach burnout inventory across small business owners and educators 显示文摘JAMES S B DWANE H D JOE M R 2000Journal of Vocational Behavior2000,56,:1
19Genome, transcriptome, and secretome analysis of wood decay fungus Postia placenta supports unique mechanisms of lignocellulose conversion 显示文摘Martinez D Challacombe J Morgenstern 1 Hibbett D Schmoll M Kubicek CP Ferreira P Ruiz-Duenas FJ Martinez AT Kersten P Hammel KE Wymelenberg AV Gaskell J Lindquist E Sabat G BonDurant SS Larrondo LF Canessa P Vicuna R Yadav J Doddapaneni H Subramanian V Pisabarro AG Lavin JL Oguiza JA Master E Henrissat B Coutinho PM Harris P Magnuson JK Baker SE Bruno K Kenealy W Hoegger PJ Kties U Ramaiya P kucas S Salamov A Shapiro H Tu H Chee CL Misra M Xie G Teter S Yaver D James T Mokrejs M Pospisek M Grigoriev IV Brettin T Rokhsar D Berka R Cullen D 2009PNAS2009,106,6:1
20COIl: an arabidopsis gene re?quired forJasmonate - regulated defense and fertility显示文摘Xie D X Feys B F James S 1998Science1998,280,5366:1
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