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| 1 | Sources and transport of algae and nutrients in a Californian river in a semi-arid climate 显示文摘 | Ohte N Dahlgren R A Silva S R | 2007 | Freshwater Biology2007，52，： | 1 |
| 2 | Models for studying long- term recovery following forebrain ischemia in the rat A 2-vessel occlusion model显示文摘 | Smith ML Bendek G Dahlgren N | 1984 | Acta Neurol Stand1984，69，： | 1 |
| 3 | Oligomeric and f ibrillar species of Amyloid-β peptides显示文摘 | Dahlgren Karie N Manelli Arlene M Stine W Blaine | 2002 | J Biol Chem2002，277，32： | 1 |
| 4 | Oligomeric and fibrillar species of amyloid-beta peptides diferentially affect neuronal viability 显示文摘 | Dahlgren K N Maneli A M Stine W B Jr | 2002 | J Biol Chem2002，277，32： | 1 |
| 5 | Genetic differences within and between species of deep sea crabs (Chaceon)from the North Atlantic Ocean显示文摘 | WEINBERG J R DAHLGREN T G TROWBRIDGE N | 2003 | Biological Bulle- tin2003，204，3： | 1 |
| 6 | Severe neurological complicationafter central neoraxial blockades in Sweden 1990-1999显示文摘 | Moen V Dahlgren N Irestedt L | 2004 | Anes-thesiology2004，101，4： | 1 |
| 7 | Models for studying longterm recovery following forebrain ischemia in the rat显示文摘 | Smith ML Bendek G Dahlgren N | 1984 | Acta Neurol Scand1984，9，6： | 1 |
| 8 | Oligomeric and fibrillar species of amyloid-beta peptides differentially affect neuronal viability显示文摘 | Dahlgren K N Manelli A M Stine W B | 2002 | J Biol Chem2002，277，32： | 1 |
| 9 | Models for studying long-term recovery following forebrain ischemia in the rat:A 2-vessel occlusion model显示文摘 | Smith M L Bendek G Dahlgren N | 1984 | Acta Neurol Scand1984，69，6： | 1 |
| 10 | Variation in vegetative and flowering phenology in a forest herb caused by environmental heterogeneity显示文摘 | Dahlgren J P von Zeipel H Ehrlén J | 2007 | American Journal of Botany2007，94，9： | 1 |
| 11 | Neurological complications after anesthesia:a follow-up of 18 000 spinal and epidural anesthetics performed over three years显示文摘 | Dahlgren N Tornebrandt K | 1995 | Acta Anaesth Scand1995，39，： | 1 |
| 12 | Severe neurological complications after central nonracial blockades in Sweden 1990-1999显示文摘 | Moen V Dahlgren N Irestedt L | 2004 | Anesthesiology2004，101，4： | 1 |
| 13 | Genetic differences within and between species of deep-sea crabs (Chaceon) from the North Atlantic Ocean显示文摘 | Weinberg J R Dahlgren N D Halanych K M | 2003 | Biological Bulletin2003，，204： | 1 |
| 14 | Models for studying long-term recovery following fore- brain ischemia in the rat :2-vessel occlusion model 显示文摘 | SMITH M I BBENDEK G DAHLGREN N | 1984 | Acta Neurol Scand1984，69，5： | 1 |
| 15 | Genetic differences within and between species of deep-sea crabs (Chaceon) from the North Atlantic Ocean 显示文摘 | Weinberg J R Dahlgren N D Halanych K M | 2003 | Biological Bulletin2003，204，： | 1 |
| 16 | Temporal variability in water quality of agricultural tailwaters: implications for water quality monitoring显示文摘 | Brauer N O Geen A T Dahlgren R A | 2009 | Agricultural Water Management2009，96，6： | 1 |
| 17 | Bone marrow mononuclear cells for joint therapy: The role of macrophages in inflammation resolution and tissue repair显示文摘Osteoarthritis(OA)is the most prevalent joint disease causing major disability and medical expenditures.Synovitis is a central feature of OA and is primarily driven by macrophages.Synovial macrophages not only drive inflammation but also its resolution,through a coordinated,simultaneous expression of pro-and anti-inflammatory mechanisms that are essential to counteract damage and recover homeostasis.Current OA therapies are largely based on anti-inflammatory principles and therefore block pro-inflammatory mechanisms such as prostaglandin E2 and Nuclear factor-kappa B signaling pathways.However,such mechanisms are also innately required for mounting a pro-resolving response,and their blockage often results in chronic low-grade inflammation.Following minor injury,macrophages shield the damaged area and drive tissue repair.If the damage is more extensive,macrophages incite inflammation recruiting more macrophages from the bone marrow to maximize tissue repair and ultimately resolve inflammation.However,sustained damage and inflammation often overwhelms pro-resolving mechanisms of synovial macrophages leading to the chronic inflammation and related tissue degeneration observed in OA.Recently,experimental and clinical studies have shown that joint injection with autologous bone marrow mononuclear cells replenishes inflamed joints with macrophage and hematopoietic progenitors,enhancing mechanisms of inflammation resolution,providing remarkable and long-lasting effects.Besides creating an ideal environment for resolution with high concentrations of interleukin-10 and anabolic growth factors,macrophage progenitors also have a direct role in tissue repair.Macrophages constitute a large part of the early granulation tissue,and further transdifferentiate from myeloid into a mesenchymal phenotype.These cells,characterized as fibrocytes,are essential for repairing osteochondral defects.Ongoing“omics”studies focused on identifying key drivers of macrophagemediated resolution of joint inflammation and those required for efficient osteochondral repair,have the potential to uncover ways for developing engineered macrophages or off-the-shelf pro-resolving therapies that can benefit patients suffering from many types of arthropaties,not only OA. | Bruno C Menarim James N MacLeod Linda A Dahlgren | 2021 | World Journal of Stem Cells2021，13，7： | 1 |
| 18 | Severe neurological complications after central neuraxial blockades in sweden 1990-1999显示文摘 | MOEN V DAHLGREN N IRESTEDT L | 2004 | Anesthesiology2004，101，4： | 1 |
| 19 | Severe neurological complications after central neuraxial blockades in Sweden 1990-1999显示文摘 | Moen V Dahlgren N Irestedt L | 2004 | Anesthesiology2004，101，4： | 1 |
| 20 | Models for studying long-term recover following forebrain ischemia in the rat: 2 A 2 vessel occlusion model显示文摘 | Smith ML Bendek G Dahlgren N | 1984 | Acta Neurol Scand1984，69，5： | 1 |
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